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Clinical trials demonstrate superior glycemic control and weight reduction compared to selective GLP1 agonists [3] [4]

The core discrepancies may stem from two factors, first, distinct genetic backgrounds of mouse strains, with inherent differences in gut microbiota responses to disease between arthritis-susceptible and conventional models, and second, undefined Clostridium strains, which fail to rule out the possibility of pathogenic subtypes

Biomedicines 9 (8), 992

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